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The EMBO Journal is a science journal containing review articles and research papers in the broad areas of molecular, structural and cellular biology.
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Stalling in mitosis and releasing the apoptotic brake

Thu, 07/22/2010 - 02:00

Stalling in mitosis and releasing the apoptotic brake

The EMBO Journal 29, 2255 (2010). doi:10.1038/emboj.2010.150

Authors: Manabu Kurokawa & Sally Kornbluth

EMBO J2914, 2407–2420 (2010); published online 4 June 2010Microtubule poisons induce mitotic arrest that leads to apoptotic cell death if not resolved in a timely manner, but the mechanisms that directly link this cell cycle arrest to apoptosis have been elusive. In this issue of The EMBO Journal, Clarke and colleagues show that Mcl-1, an anti-apoptotic Bcl-2 family protein, is phosphorylated by the mitotic kinase CDK1/cyclin B1. This targets the protein for degradation by anaphase-promoting complex/cyclosome (APC/C)-mediated ubiquitination, in a manner such that only prolonged arrest allows sufficient Mcl-1 phosphorylation and degradation to trigger apoptosis. Thus, the APC/C, a major effector of the spindle assembly checkpoint (SAC), not only ensures cell cycle arrest upon spindle disruption, but promotes cell death when the duration of mitotic arrest is too long.

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Cell polarity/motility in bacteria: closer to eukaryotes than expected?

Thu, 07/22/2010 - 02:00

Cell polarity/motility in bacteria: closer to eukaryotes than expected?

The EMBO Journal 29, 2258 (2010). doi:10.1038/emboj.2010.144

Author: Emilia M F Mauriello

EMBO J2914, 2276–2289 (2010); published online 11 June 2010The Gram-negative bacterium Myxococcus xanthus glides on solid surfaces and periodically reverses the direction of movement. Work published in this issue of The EMBO Journal (Leonardy et al, 2010) reports on the small GTPase MglA that ensures the correct polarity of the motility engines through its GTP/GDP cycle in conjunction with its cognate GAP, MglB. MglA has also been shown to interact with the actin-like protein MreB in eukaryotic-like motility complexes. Altogether, the data suggest that compelling similarities exist between the mechanisms of motility and establishment of cell polarity in M. xanthus and eukaryotes.

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Twins take the job

Thu, 07/22/2010 - 02:00

Twins take the job

The EMBO Journal 29, 2260 (2010). doi:10.1038/emboj.2010.148

Authors: María-Eugenia Gas & Bertrand Séraphin

EMBO J2914, 2358–2367 (2010); published online 8 June 2010EMBO J2914, 2342–2357 (2010); published online 8 June 2010The exosome, one of the main cellular ribonucleases in eukaryotes, is a multi-subunit complex of deep evolutionary origin. This complex has been extensively characterized in Saccharomyces cerevisae. Given the good conservation of exosome subunit sequences, it was widely accepted that the yeast exosome provided a good model for other eukaryotic species. Recent analysis of higher eukaryotic exosomes reveals some differences challenging this idea. In this issue of The EMBO Journal, Tomecki et al (2010) and Staals et al (2010) provide new insights into the composition and function of the human exosome, with implications for our understanding on the organization, function and localization of this complex in these cells.

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Channels get in an HUFA: RNA editing gets them out of a jam

Thu, 07/08/2010 - 02:00

Channels get in an HUFA: RNA editing gets them out of a jam

The EMBO Journal 29, 2097 (2010). doi:10.1038/emboj.2010.134

Authors: Richard Horn & Robert Reenan

EMBO J2913, 2101–2113 (2010); published online 11 May 2010The enzymatic conversion of adenosine-to-inosine (A-to-I) in RNA rewrites the informational output of a number of different classes of coding and non-coding RNA transcripts. In this issue, Decher et al (2010) reveal a new functional consequence for an ancient RNA editing site in certain animal voltage-gated K+ channel (Kv) genes. Abundant highly unsaturated fatty acids (HUFAs), which are essential in mammals for normal brain function, block conduction of Kv channels—an effect that can be nearly obliterated by the recoding of a single amino acid through RNA editing.

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Receptor endocytosis: Frizzled joins the ubiquitin club

Thu, 07/08/2010 - 02:00

Receptor endocytosis: Frizzled joins the ubiquitin club

The EMBO Journal 29, 2099 (2010). doi:10.1038/emboj.2010.132

Author: Ken M Cadigan

EMBO J2913, 2114–2125 (2010); published online 14 May 2010Wnt/β-catenin signalling is initiated by binding of secreted Wnt ligands to Frizzled and LRP5/6/Arrow co-receptors. A new study in this issue of The EMBO Journal provides compelling evidence that the level of cell surface Frizzled is controlled by a cycle of mono-ubiquitylation–deubiquitylation, the latter being mediated by the deubiquitylating enzyme UBPY/USP8. The amount of Frizzled on the plasma membrane appears to be a major rate-limiting factor in determining a cell's Wnt responsiveness.

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The NoRC complex mediates heterochromatin formation and stability of silent rRNA genes and centromeric repeats

Thu, 07/08/2010 - 02:00

The NoRC complex mediates heterochromatin formation and stability of silent rRNA genes and centromeric repeats

The EMBO Journal 29, 2253 (2010). doi:10.1038/emboj.2010.145

Authors: Claudio Guetg, Philipp Lienemann, Valentina Sirri, Ingrid Grummt, Danièle Hernandez-Verdun, Michael O Hottiger, Martin Fussenegger & Raffaella Santoro

Correction to:The EMBO Journal (2010) 29, 2135–2146. doi:10.1038/emboj.2010.17; Published online 18 February 2010The author list of the online version of the above paper by Guetg et al was incorrect. Martin Fussenegger's authorship was

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Crystal structure of the yeast Sac1: implications for its phosphoinositide phosphatase function

Thu, 07/01/2010 - 23:00

Crystal structure of the yeast Sac1: implications for its phosphoinositide phosphatase function

The EMBO Journal 29, 2472 (2010). doi:10.1038/emboj.2010.146

Authors: Andrew Manford, Tian Xia, Ajay Kumar Saxena, Christopher Stefan, Fenghua Hu, Scott D Emr & Yuxin Mao

Correction to: The EMBO Journal (2010) 29, 1489–1498; doi:10.1038/emboj.2010.57In the above article, the authors omitted the current affiliation of Dr Ajay Kumar Saxena, which is: Structural Biology Lab, School of Life Sciences, Jawaharlal Nehru University,

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P2X4 receptors mediate PGE2 release by tissue-resident macrophages and initiate inflammatory pain

Thu, 06/17/2010 - 23:00

P2X4 receptors mediate PGE2 release by tissue-resident macrophages and initiate inflammatory pain

The EMBO Journal 29, 2290 (2010). doi:10.1038/emboj.2010.126

Authors: Lauriane Ulmann, Hélène Hirbec & François Rassendren

Prostaglandin E2 (PGE2) is a key mediator of inflammation and contributes to pain hypersensitivity by promoting sensory neurons hyperexcitability. PGE2 synthesis results from activation of a multi-step enzymatic cascade that includes cyclooxygenases (COXs), the main targets of non-steroidal anti-inflammatory drugs (NSAIDs). Although NSAIDs are widely

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Cooperative interactions at the SLP-76 complex are critical for actin polymerization

Thu, 06/17/2010 - 23:00

Cooperative interactions at the SLP-76 complex are critical for actin polymerization

The EMBO Journal 29, 2315 (2010). doi:10.1038/emboj.2010.133

Authors: Mira Barda-Saad, Naoto Shirasu, Maor H Pauker, Nirit Hassan, Orly Perl, Andrea Balbo, Hiroshi Yamaguchi, Jon C D Houtman, Ettore Appella, Peter Schuck & Lawrence E Samelson

T-cell antigen receptor (TCR) engagement induces formation of multi-protein signalling complexes essential for regulating T-cell functions. Generation of a complex of SLP-76, Nck and VAV1 is crucial for regulation of the actin machinery. We define the composition, stoichiometry and specificity of interactions in the SLP-76,

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The ATAC acetyl transferase complex controls mitotic progression by targeting non-histone substrates

Thu, 06/17/2010 - 23:00

The ATAC acetyl transferase complex controls mitotic progression by targeting non-histone substrates

The EMBO Journal 29, 2381 (2010). doi:10.1038/emboj.2010.125

Authors: Meritxell Orpinell, Marjorie Fournier, Anne Riss, Zita Nagy, Arnaud R Krebs, Mattia Frontini & Làszlò Tora

All DNA-related processes rely on the degree of chromatin compaction. The highest level of chromatin condensation accompanies transition to mitosis, central for cell cycle progression. Covalent modifications of histones, mainly deacetylation, have been implicated in this transition, which also involves transcriptional repression. Here, we show

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CaMKII hunkers down on the muscarinic M4 receptor to help curb cocaine-induced hyperlocomotion

Thu, 06/17/2010 - 02:00

CaMKII hunkers down on the muscarinic M4 receptor to help curb cocaine-induced hyperlocomotion

The EMBO Journal 29, 1943 (2010). doi:10.1038/emboj.2010.105

Authors: Ivar S Stein & Johannes W Hell

EMBO J2912, 2070–2081 (2010); published online 11 May 2010The dopaminergic receptor D1 activates and the muscarinic acetylcholine receptor (mAChR) M4 inhibits adenylyl cyclase (AC) and thereby cAMP production through the trimeric Gαs and Gαi/o proteins, respectively (Wess et al, 2007). D1 and M4 are coexpressed in striatal output neurons, where M4 antagonizes D1 signalling as recently pinpointed by conditional M4 knockout in D1-expressing neurons (Jeon et al, 2010). Closely following on the heels of this article, Guo et al (2010) report in this issue that CaMKII binds and phosphorylates M4 upon Ca2+ influx to augment the antagonistic action of M4 on D1 signalling and on D1-mediated, cocaine-triggered hyperlocomotion.

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How do tumours adapt to nutrient stress?

Thu, 06/17/2010 - 02:00

How do tumours adapt to nutrient stress?

The EMBO Journal 29, 1946 (2010). doi:10.1038/emboj.2010.110

Authors: Ronald C Wek & Kirk A Staschke

EMBO J2912, 2082–2096 (2010); published online 14 May 2010Reduced blood flow contributes to tumour hypoxia and nutrient deprivation forcing cancer cells to adapt to ensure survival. In this issue of The EMBO Journal, Ye et al show that a stress response pathway including the eIF2 kinase GCN2 and its downstream target, the transcriptional activator ATF4, is critical for proliferation and survival of tumour cells after starvation for amino acids or glucose and is essential for growth in vivo in a xenograft model. This study provides new mechanistic insight into how tumour cells sense and adapt to nutrient restriction and suggests new approaches for cancer chemotherapy.

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A classical NLS and the SUN domain contribute to the targeting of SUN2 to the inner nuclear membrane

Mon, 06/14/2010 - 23:00

A classical NLS and the SUN domain contribute to the targeting of SUN2 to the inner nuclear membrane

The EMBO Journal 29, 2262 (2010). doi:10.1038/emboj.2010.119

Authors: Yagmur Turgay, Rosemarie Ungricht, Andrea Rothballer, Alexa Kiss, Gabor Csucs, Peter Horvath & Ulrike Kutay

Integral membrane proteins of the inner nuclear membrane (INM) are inserted into the endoplasmic reticulum membrane during their biogenesis and are then targeted to their final destination. We have used human SUN2 to delineate features that are required for INM targeting and have identified multiple

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Ezrin tunes T-cell activation by controlling Dlg1 and microtubule positioning at the immunological synapse

Mon, 06/14/2010 - 23:00

Ezrin tunes T-cell activation by controlling Dlg1 and microtubule positioning at the immunological synapse

The EMBO Journal 29, 2301 (2010). doi:10.1038/emboj.2010.127

Authors: Rémi Lasserre, Stéphanie Charrin, Céline Cuche, Anne Danckaert, Maria-Isabel Thoulouze, Fabrice de Chaumont, Tarn Duong, Nathalie Perrault, Nadine Varin-Blank, Jean-Christophe Olivo-Marin, Sandrine Etienne-Manneville, Monique Arpin, Vincenzo Di Bartolo & Andrés Alcover

T-cell receptor (TCR) signalling is triggered and tuned at immunological synapses by the generation of signalling complexes that associate into dynamic microclusters. Microcluster movement is necessary to tune TCR signalling, but the molecular mechanism involved remains poorly known. We show here that the membrane-microfilament linker

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TCR-mediated ThPOK induction promotes development of mature (CD24−) γδ thymocytes

Mon, 06/14/2010 - 23:00

TCR-mediated ThPOK induction promotes development of mature (CD24−) γδ thymocytes

The EMBO Journal 29, 2329 (2010). doi:10.1038/emboj.2010.113

Authors: Kyewon Park, Xi He, Hyung-Ok Lee, Xiang Hua, Yi Li, David Wiest & Dietmar J Kappes

T lymphocytes develop into two major lineages characterized by expression of the αβ and γδ T cell receptor (TCR) heterodimers. Within each major lineage, further specialization occurs, resulting in distinct subsets that differ in TCR specificity, phenotype and functional attributes. Thus, in the murine thymus,

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CIN85 regulates dopamine receptor endocytosis and governs behaviour in mice

Mon, 06/14/2010 - 23:00

CIN85 regulates dopamine receptor endocytosis and governs behaviour in mice

The EMBO Journal 29, 2421 (2010). doi:10.1038/emboj.2010.120

Authors: Noriaki Shimokawa, Kaisa Haglund, Sabine M Hölter, Caroline Grabbe, Vladimir Kirkin, Noriyuki Koibuchi, Christian Schultz, Jan Rozman, Daniela Hoeller, Chun-Hong Qiu, Marina B Londoño, Jun Ikezawa, Peter Jedlicka, Birgit Stein, Stephan W Schwarzacher, David P Wolfer, Nicole Ehrhardt, Rainer Heuchel, Ioannis Nezis, Andreas Brech, Mirko H H Schmidt, Helmut Fuchs, Valerie Gailus-Durner, Martin Klingenspor, Oliver Bogler, Wolfgang Wurst, Thomas Deller, Martin Hrabé de Angelis & Ivan Dikic

Despite extensive investigations of Cbl-interacting protein of 85 kDa (CIN85) in receptor trafficking and cytoskeletal dynamics, little is known about its functions in vivo. Here, we report the study of a mouse deficient of the two CIN85 isoforms expressed in the central nervous system,

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SNMIB/Apollo protects leading-strand telomeres against NHEJ-mediated repair

Mon, 06/14/2010 - 23:00

SNMIB/Apollo protects leading-strand telomeres against NHEJ-mediated repair

The EMBO Journal 29, 2230 (2010). doi:10.1038/emboj.2010.58

Authors: Yung C Lam, Shamima Akhter, Peili Gu, Jing Ye, Anaïs Poulet, Marie-Josèphe Giraud-Panis, Susan M Bailey, Eric Gilson, Randy J Legerski & Sandy Chang

Progressive telomere attrition or deficiency of the protective shelterin complex elicits a DNA damage response as a result of a cell's inability to distinguish dysfunctional telomeric ends from DNA double-strand breaks. SNMIB/Apollo is a shelterin-associated protein and a member of the SMN1/PSO2 nuclease family that

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Regulation of dynamic polarity switching in bacteria by a Ras-like G-protein and its cognate GAP

Thu, 06/10/2010 - 23:00

Regulation of dynamic polarity switching in bacteria by a Ras-like G-protein and its cognate GAP

The EMBO Journal 29, 2276 (2010). doi:10.1038/emboj.2010.114

Authors: Simone Leonardy, Mandy Miertzschke, Iryna Bulyha, Eva Sperling, Alfred Wittinghofer & Lotte Søgaard-Andersen

The rod-shaped cells of the bacterium Myxococcus xanthus move uni-directionally and occasionally undergo reversals during which the leading/lagging polarity axis is inverted. Cellular reversals depend on pole-to-pole relocation of motility proteins that localize to the cell poles between reversals. We show that MglA is

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The C-terminal α–α superhelix of Pat is required for mRNA decapping in metazoa

Thu, 06/10/2010 - 23:00

The C-terminal α–α superhelix of Pat is required for mRNA decapping in metazoa

The EMBO Journal 29, 2368 (2010). doi:10.1038/emboj.2010.124

Authors: Joerg E Braun, Felix Tritschler, Gabrielle Haas, Cátia Igreja, Vincent Truffault, Oliver Weichenrieder & Elisa Izaurralde

Pat proteins regulate the transition of mRNAs from a state that is translationally active to one that is repressed, committing targeted mRNAs to degradation. Pat proteins contain a conserved N-terminal sequence, a proline-rich region, a Mid domain and a C-terminal domain (Pat-C). We show that

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Structural insights into the exquisite selectivity of neurexin/neuroligin synaptic interactions

Thu, 06/10/2010 - 23:00

Structural insights into the exquisite selectivity of neurexin/neuroligin synaptic interactions

The EMBO Journal 29, 2461 (2010). doi:10.1038/emboj.2010.123

Authors: Philippe Leone, Davide Comoletti, Géraldine Ferracci, Sandrine Conrod, Simon U Garcia, Palmer Taylor, Yves Bourne & Pascale Marchot

The extracellular domains of neuroligins and neurexins interact through Ca2+ to form flexible trans-synaptic associations characterized by selectivity for neuroligin or neurexin subtypes. This heterophilic interaction, essential for synaptic maturation and differentiation, is regulated by gene selection, alternative mRNA splicing and post-translational

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